How Many Complications Are There for Gout?
1. Acute kidney injury: The deposition of uric acid crystals in the kidneys can lead to acute obstructive nephropathy or interstitial nephritis;
2. Chronic renal insufficiency: Long-term hyperuricemia can lead to chronic interstitial nephritis and hypertension, ultimately progressing to chronic renal failure;
3. Urolithiasis: Due to the increased concentration of uric acid in urine, uric acid salts are prone to form crystals and deposit in various parts of the urinary system, such as the ureter, bladder, and urethra, causing uric acid stones.
1. Atherosclerosis: Numerous studies have shown that hyperuricemia is one of the important risk factors for the development of coronary heart disease, which is closely related to lipid disorders;
2. Hypertension: Gout patients often have obesity, impaired glucose tolerance, diabetes, and other issues, which all increase the risk of hypertension;
3. Left ventricular hypertrophy: Studies have shown that the thickness of the left ventricular wall of gout patients is significantly higher than that of non-gout patients, suggesting that it may be related to high uric acid levels.
When gout attacks, if it involves the cranial nerves, it may lead to increased intracranial pressure, causing symptoms such as headache and vomiting, and severe cases may even lead to coma.
After gout tophi erode soft tissues, they can cause joint destruction, thereby affecting limb movements and causing joint deformities, such as inward or outward curvature of the toes.
In addition to the above situations, some patients may also experience skin damage, fundus lesions, and other conditions. Therefore, it is recommended that people with gout promptly visit the rheumatology and immunology department of the hospital and actively cooperate with doctors for treatment to avoid delaying the condition.
