Generally, if a patient has mild or moderate gout and actively undergoes standardized drug treatment, it usually does not cause severe damage to the body. However, if the condition persists and is not controlled promptly, it may lead to joint destruction and renal dysfunction.
1. Joint Destruction:
As the disease progresses, if urate crystals deposit and gradually increase in the joints, they can cause chronic inflammatory reactions, resulting in joint pain, swelling, and other symptoms. If not intervened in time, this can lead to erosion of joint cartilage, ultimately causing permanent structural changes.
2. Renal Damage:
Long-term hyperuricemia can easily induce hypertension and cardiovascular and cerebrovascular diseases, which can also affect the kidneys during this process, causing abnormal kidney metabolism and leading to the formation of tophus, and even increasing the risk of kidney stones. Additionally, the inability to normally excrete purine substances from the body may also affect liver and kidney function, further impacting overall health. Therefore, it is recommended that after being diagnosed with gout, patients should actively cooperate with doctors in standardized drug treatment, such as febuxostat tablets, allopurinol sustained-release capsules, and other drugs that inhibit uric acid synthesis; benzbromarone tablets, probenecid tablets, and other drugs that promote uric acid excretion. Daily life should also involve a reasonable diet, avoiding purine-rich foods such as seafood and animal liver, which may hinder disease recovery.