What are the Benefits and Effects of Huangqi Injection?

Update Date: Source: Network

Huangqi Injection has the effects of nourishing the heart and unblocking the pulse, invigorating Qi and nourishing the origin, strengthening the spleen and removing dampness, as well as strengthening the body and dispelling evils. In clinical practice, it is mainly used to treat nephritis, nephrotic syndrome, myocarditis, hepatitis, cardiac insufficiency, spleen deficiency, and other diseases. It has a good adjuvant therapeutic effect on Qi deficiency diseases caused by various reasons, and it can also increase the number of white blood cells in the human body, which has a certain effect on enhancing the body's immunity.

Efficacy 1:

It has anti-myocardial ischemia and myocardial protective effects, which can reduce the left ventricular dilation and infarct area extension in myocardial infarction model rats, improve left ventricular contraction and relaxation functions, and improve left ventricular remodeling after myocardial infarction. It can clear free radicals and improve left ventricular diastolic and systolic functions. It can increase coronary flow, myocardial contraction amplitude, and heart rate in normal isolated rat hearts, and increase the activity of Na+, K+-ATPase on myocardial cell membranes.

Efficacy 2:

It has protective effects on the kidneys, which can reduce the levels of endothelin-1 and malondialdehyde (MDA), a lipid peroxidation product, in the plasma of rats with renal ischemia-reperfusion injury, increase the activity of superoxide dismutase in the plasma, and slow down the thickening of the glomerular basement membrane.

Efficacy 3:

It has protective effects on the liver. Huangqi Injection can improve the activities of alanine aminotransferase and glutathione S-transferase in the serum of rats with intestinal ischemia-reperfusion-induced liver injury, as well as the content of MDA and SOD activity in liver tissue.

Efficacy 4:

It has protective effects on the lungs. It can reduce the levels of xanthine oxidase and MDA in rabbits with superior mesenteric artery occlusion-induced shock lung injury, reduce the production of lipid peroxide (LPO), and partially inhibit the occurrence of hypoxic pulmonary hypertension.